What a working Ξ²-catenin inhibitor would be worth to the United States
A transparent GCEA estimate of the lifetime societal value of an effective Wnt/Ξ²-catenin:TCF inhibitor β modeled on the clinical premise of Parabilis Medicines' zolucatetide. Every assumption below is editable; the answer recomputes live. This is a societal-value question, not a sales forecast or company valuation.
Year-1 value by engine
intermediate subtotalsCumulative discounted value over time
PV accrualWhere the value comes from
70-yr PV by indicationEngine A β Chronic lifelong maintenance
desmoid Β· FAP Β· ACP| Indication | Prevalent pool | Treated % | Averted med $/pt/yr | QoL+prod $/pt/yr | Yr-1 value | 70-yr PV |
|---|---|---|---|---|---|---|
| Subtotal |
Engine B β Oncology survival
per-responder lifetime value| Indication | Annual incidence | Address % | Benefit % | LY gained | Avert $k | Prod $k | Care $k | Yr-1 value | 70-yr PV |
|---|---|---|---|---|---|---|---|---|---|
| Subtotal |
Engine C β Immune resensitization
layer OFF| Conversion target | Annual incidence | Conversion % | Durable LY | Avert $k | Prod $k | Care $k | Yr-1 value | 70-yr PV |
|---|---|---|---|---|---|---|---|---|
| Subtotal |
One-way sensitivity β CRC
benefit % Γ LY gained β 70-yr PVMethodology & notes
show your workThis model follows the Generalized Cost-Effectiveness Analysis (GCEA) framework of Shafrin et al., "Valuing the Societal Impact of Medicines and Other Health Technologies: A User Guide to Current Best Practices," Forum for Health Economics & Policy (2024). It applies the guide's recommended choices: a 2% societal discount rate, 70 stacked annual cohorts over a 70-year horizon, dynamic net health system costs via genericization, and dynamic prevalence through population growth and aging.
Two value engines
Engine A β chronic lifelong maintenance (desmoid, FAP, and adamantinomatous craniopharyngioma [ACP] β a near-uniformly CTNNB1/Ξ²-catenin-driven tumor managed long-term). A prevalent pool is treated continuously; value recurs every year for life. The pool grows with population only.
Engine B β oncology survival (CRC, HCC, rare low-complexity, and a broad "other Ξ²-catenin" bucket). Each responder gets a one-time lifetime value; the count of new responders grows with population and aging.
Engine C β immune resensitization (optional, additive). Conversions of IO-refractory tumors into durable responders; durable LY set above the cytostatic base case because durable IO responses behave like functional cures.
Integration & discounting
Each stream is computed year by year for t = 0β¦Hβ1, compounded at its own real growth rate, then discounted at the societal rate: PV = Ξ£ flow_t / (1+r)^t. The post-generic share is the fraction of PV arriving in year 14 and later β the value that accrues to society after the drug's own price has collapsed toward marginal cost.
Growth, with taper (not flat for 70 years)
Population growth starts at the slider value and tapers linearly to zero by ~year 50. The aging uplift to cancer incidence starts at its slider value, holds through ~year 15, then fades to zero by ~year 40. Real medical-cost growth applies only to averted-medical-cost terms, so averted hospitalizations and surgeries are worth more later; VOLY and productivity are held flat in real terms.
Drug cost as a transfer
For a societal-value estimate the drug's own acquisition cost is treated as a transfer β 0 and excluded. The drug is branded ~14 years, then its price collapses toward marginal cost; societal value persists after that. The model therefore reports how much of the total value lands in the post-generic period.
GCEA petals: included vs. conservatively omitted
The guide treats every omitted petal as real value, so excluding them biases this estimate downward. They are upside left on the table.